CB2 Cannabinoid Receptor Expressing HEK293 Cell Lines
Human, mouse or rat cannabinoid receptor type 2 (CB2) expressing HEK293 cells.
CB2 cannabinoid receptors are a component of the endocannabinoid system. While not as widespread as CB1 receptors, CB2 receptors are highly inducible following tissue injury and may represent novel therapeutic targets for a variety of pathological conditions. In healthy organisms, CB2 receptors ere expressed mainly in the immune system and in cell of hematopoietic origin (e.g., microglia).
From the laboratory of Ken Mackie, MD, Indiana University.
Human, mouse or rat cannabinoid receptor type 2 (CB2) expressing HEK293 cells.
CB2 cannabinoid receptors are a component of the endocannabinoid system. While not as widespread as CB1 receptors, CB2 receptors are highly inducible following tissue injury and may represent novel therapeutic targets for a variety of pathological conditions. In healthy organisms, CB2 receptors ere expressed mainly in the immune system and in cell of hematopoietic origin (e.g., microglia).
From the laboratory of Ken Mackie, MD, Indiana University.
| Catalog Number | Product | DataSheet | Size | AVAILABILITY | Price | Qty |
|---|
Specifications
| Product Type: | Cell Line |
| Cell Type: | HEK293 |
| Accession ID: | P34972 |
| Organism: | Human, mouse or rat |
| Source: | Embryonic Kidney |
| Morphology: | Epithelial |
| Biosafety Level: | BSL-2 |
| Growth Conditions: |
DMEM, 10% FBS, (1mg/mL) G418, Pen/Strep (See  Suggested Protocol for details)
|
| Subculturing: | 0.05% Trypsin-EDTA/Versene |
| Cryopreservation: | 90% FBS, 10% DMSO |
| Comments: | CB2 receptor has a N-terminus HA tag |
| Storage: | Liquid nitrogen |
| Shipped: | Dry Ice |
Documentation
Provider
From the laboratory of Ken Mackie, MD, Indina University.
References
- Atwood BK, Wager-Miller J, Haskins C, Straiker A, Mackie K. Functional selectivity in CB(2) cannabinoid receptor signaling and regulation: implications for the therapeutic potential of CB(2) ligands. Mol Pharmacol. 2012 Feb;81(2):250-63.
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