Anti-Golgin-97 [CDF4] Antibody

This mouse IgG1 monoclonal antibody [CDF4] was generated against recombinant protein and recognizes human and rabbit Golgin-97.

Highlights:

  • Reacts with human and rabbit Golgin-97
  • Suitable for Western Blot and Immunofluorscence applications

The GRIP family member, golgin 97, is a trans-Golgi network peripheral membrane protein that resides on the cytoplasmic face of the Golgi. Binding of golgin 97 to the Golgi membrane is mediated by the G protein family member, Arl1. Golgin 97 plays an essential role to the cell in the form of a tethering molecule associating with tubulovesicular carriers during the trafficking from the trans-Golgi network to the recycling endosome and/or early endosome.

From the laboratory of Marvin J. Fritzler, MD, PhD, University of Calgary.

The Investigator's Annexe Part of The Investigator's Annexe program.

Catalog Number Product DataSheet Size AVAILABILITY Price Qty
EUC009
Anti-Golgin-97 [CDF4] Antibody
1mL In stock
Regular Price:$285.00
On Sale:
Specifications

Product Type: Antibody
Alternative Name(s): Golgin subfamily A member 1
Antigen: Golgin 97
Accession ID: Q92805, Q96JQ5
Molecular Weight: 88 kDa
Isotype: IgG1
Clonality: Monoclonal
Clone Name: CDF4
Reactivity: Human and rabbit
Immunogen: Mouse cell line 300-19 expressing transfected human MS4A4A-GFP
Species Immunized: Mouse
Buffer: Cell culture supernatant
Tested Applications: IF 1:200-1:1000, WB 1:100-1:500
Storage: -80C
Shipped: Cold Packs

Provider
From the laboratory of Marvin J. Fritzler, MD, PhD, University of Calgary.
References
  1. KJ Griffith, EKL Chan, JC Hamel, K Miyachi, MJ Fritzler. Molecular characterization of a novel 97 kDa Golgi complex autoantigen recognized by autoimmune antibodies from patients with Sjögren's syndrome. Arthritis Rheum. 40:1693-1702, 1997.
  2. K Nozawa, M J Fritzler, Y Takasaki, MR Wood, EKL. Chan. Co-clustering of Golgi complex and other cytoplasmic organelles to crescentic region of half-moon nuclei during apoptosis. Cell Biol.Int. 33:148-157, 2009.

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